Tumor necrosis factor α increases neuronal vulnerability to excitotoxic necrosis by inducing expression of the AMPA-glutamate receptor subunit GluR1 via an acid sphingomyelinase-and NF-κB-dependent mechanism

ZaiFang Yu, Guanjun Cheng, Xiaoming Wen, Gary D. Wu, Wang Tso Lee, David E Pleasure

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Medicine & Life Sciences