Stabilizing and Remodeling the Blocked DNA Replication Fork: Anchoring FANCM and the Fanconi Anemia Damage Response

Larry H. Thompson; Nigel J. Jones

doi:10.1016/j.molcel.2010.03.003

Stabilizing and Remodeling the Blocked DNA Replication Fork: Anchoring FANCM and the Fanconi Anemia Damage Response

Larry H. Thompson, Nigel J. Jones

Research output: Contribution to journal › Article › peer-review

6 Scopus citations

Abstract

In this issue of Molecular Cell, Yan et al. (2010) and Singh et al. (2010) identify an evolutionarily conserved FANCM-associated histone-fold MHF heterodimer (MHF1-MHF2) that promotes the remodeling of artificial replication forks and confers cellular resistance to DNA crosslinks and camptothecin.

Original language	English (US)
Pages (from-to)	749-751
Number of pages	3
Journal	Molecular Cell
Volume	37
Issue number	6
DOIs	https://doi.org/10.1016/j.molcel.2010.03.003
State	Published - Mar 26 2010
Externally published	Yes

ASJC Scopus subject areas

Molecular Biology
Cell Biology

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abstract = "In this issue of Molecular Cell, Yan et al. (2010) and Singh et al. (2010) identify an evolutionarily conserved FANCM-associated histone-fold MHF heterodimer (MHF1-MHF2) that promotes the remodeling of artificial replication forks and confers cellular resistance to DNA crosslinks and camptothecin.",

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AU - Jones, Nigel J.

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AB - In this issue of Molecular Cell, Yan et al. (2010) and Singh et al. (2010) identify an evolutionarily conserved FANCM-associated histone-fold MHF heterodimer (MHF1-MHF2) that promotes the remodeling of artificial replication forks and confers cellular resistance to DNA crosslinks and camptothecin.

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Stabilizing and Remodeling the Blocked DNA Replication Fork: Anchoring FANCM and the Fanconi Anemia Damage Response

Abstract

ASJC Scopus subject areas

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