Our recent work suggests so-called ‘foot-drop’ post-stroke results from dysfunction of dynamic hip-knee coupling during swing rather than impaired dorsiflexion. Here our aim was to examine the mutability of hip-knee coupling to identify possible mechanisms of gait recovery following therapeutic intervention. By investigating joint powers and muscle activation patterns our results revealed improvements including increased power production from the hip and knee, but not the ankle. Further, activation patterns for hip muscles demonstrated reorganization resembling controls. Taken together, these findings suggest the notion of foot-drop post-stroke should be reconsidered and the focus of treatment for hemiparetic walking dysfunction be redirected.