Effectiveness of sotalol treatment in symptomatic Brugada syndrome

Kathryn A. Glatter; Qing Wang; Mark Keating; Shenghan Chen; Nipavan Chiamvimonvat; Melvin M. Scheinman

doi:10.1016/j.amjcard.2004.02.023

Effectiveness of sotalol treatment in symptomatic Brugada syndrome

Kathryn A. Glatter, Qing Wang, Mark Keating, Shenghan Chen, Nipavan Chiamvimonvat, Melvin M. Scheinman

Cardiology (Davis)

Research output: Contribution to journal › Article › peer-review

16 Scopus citations

Abstract

We describe a 53-year-old man with recurrent syncopal events and a malignant family history who was treated for 13 years with sotalol drug therapy with no further occurrence of Brugada syndrome symptoms. Genetic testing revealed that he carried a Brugada syndrome sodium channel SCN5A mutation (4189delT). This finding suggests that sotalol may be of therapeutic benefit in such patients.

Original language	English (US)
Pages (from-to)	1320-1322
Number of pages	3
Journal	American Journal of Cardiology
Volume	93
Issue number	10
DOIs	https://doi.org/10.1016/j.amjcard.2004.02.023
State	Published - May 15 2004

ASJC Scopus subject areas

Cardiology and Cardiovascular Medicine

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title = "Effectiveness of sotalol treatment in symptomatic Brugada syndrome",

abstract = "We describe a 53-year-old man with recurrent syncopal events and a malignant family history who was treated for 13 years with sotalol drug therapy with no further occurrence of Brugada syndrome symptoms. Genetic testing revealed that he carried a Brugada syndrome sodium channel SCN5A mutation (4189delT). This finding suggests that sotalol may be of therapeutic benefit in such patients.",

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AU - Glatter, Kathryn A.

AU - Wang, Qing

AU - Keating, Mark

AU - Chen, Shenghan

AU - Chiamvimonvat, Nipavan

AU - Scheinman, Melvin M.

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N2 - We describe a 53-year-old man with recurrent syncopal events and a malignant family history who was treated for 13 years with sotalol drug therapy with no further occurrence of Brugada syndrome symptoms. Genetic testing revealed that he carried a Brugada syndrome sodium channel SCN5A mutation (4189delT). This finding suggests that sotalol may be of therapeutic benefit in such patients.

AB - We describe a 53-year-old man with recurrent syncopal events and a malignant family history who was treated for 13 years with sotalol drug therapy with no further occurrence of Brugada syndrome symptoms. Genetic testing revealed that he carried a Brugada syndrome sodium channel SCN5A mutation (4189delT). This finding suggests that sotalol may be of therapeutic benefit in such patients.

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