Corrigendum to “Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's disease” [EBioMedicine 2 (2015) 294–305] (Modulation of Mitochondrial Complex I Activity Averts Cognitive Decline in Multiple Animal Models of Familial Alzheimer's Disease (2015) 2(4) (294–305), (S2352396415000729), (10.1016/j.ebiom.2015.03.009))

Liang Zhang, Song Zhang, Izumi Maezawa, Sergey Trushin, Paras Minhas, Matthew Pinto, Lee-Way Jin, Keshar Prasain, Thi D.T. Nguyen, Yu Yamazaki, Takahisa Kanekiyo, Guojun Bu, Benjamin Gateno, Kyeong Ok Chang, Karl A. Nath, Emirhan Nemutlu, Petras Dzeja, Yuan Ping Pang, Duy H. Hua, Eugenia Trushina

Research output: Contribution to journalComment/debate

Abstract

In the version of this article originally published, in Supplementary Tables 1 and 2 CP2 concentrations were incorrectly presented in μM instead of nM. Tables are included here to reflect correct values including additional data for independent concentration evaluation. The mistake was introduced due to an error in the concentration of the standard used for the HPLC quantification. Additionally, in the Results section, the sentence “Average concentration of CP2 in the brain of new-born mice was ~90 μM, and in adult mice ~130 μM …” should be replaced with: “Average concentration of CP2 in the brain of new-born mice was ~90 nM, and in adult mice ~130 nM …”. Since CP2 was used as a model compound for a proof of concept studies, this mistake will not impact any preclinical developments.

Original languageEnglish (US)
JournalEBioMedicine
DOIs
StatePublished - Jan 1 2019

ASJC Scopus subject areas

  • Biochemistry, Genetics and Molecular Biology(all)

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