Abstract
The cardiovascular effects of equipoxent (minimum alveolar concentration) doses of halothane versus halothane plus 25% N 2O (H25N 2O) in spontaneously breathing dogs do not differ except that nitrous oxide increased mean arterial pressure (AP) and decreased arterial oxygen partial pressure (Pao 2). When 75% nitrous oxide was added to halothane anesthesia, AP, mean pulmonary artery pressure, heart rate, cardiac output, stroke volume, total peripheral resistance and left ventricular work increased and Pao 2 and hemoglobin saturation decreased. Arterial oxygen tensions below 80 torr were common at moderate and deep anesthetic levels of halothane plus 75% N 2O (H75N 2O). The specific contribution of N 2O, hypoxemia, hypercapnia, or temporal recovery (or a combination of these) in producing cardiovascular stimulation were not determined.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 197-200 |
| Number of pages | 4 |
| Journal | American Journal of Veterinary Research |
| Volume | 36 |
| Issue number | 2 |
| State | Published - Jan 1 1975 |
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- veterinary(all)
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Circulatory effects of halothane and halothane nitrous oxide anesthesia in the dog : spontaneous ventilation. / Steffey, Eugene; Gillespie, J. R.; Berry, J. D.; Eger, E. I.; Rhode, E. A.
In: American Journal of Veterinary Research, Vol. 36, No. 2, 01.01.1975, p. 197-200.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - Circulatory effects of halothane and halothane nitrous oxide anesthesia in the dog
T2 - spontaneous ventilation
AU - Steffey, Eugene
AU - Gillespie, J. R.
AU - Berry, J. D.
AU - Eger, E. I.
AU - Rhode, E. A.
PY - 1975/1/1
Y1 - 1975/1/1
N2 - The cardiovascular effects of equipoxent (minimum alveolar concentration) doses of halothane versus halothane plus 25% N 2O (H25N 2O) in spontaneously breathing dogs do not differ except that nitrous oxide increased mean arterial pressure (AP) and decreased arterial oxygen partial pressure (Pao 2). When 75% nitrous oxide was added to halothane anesthesia, AP, mean pulmonary artery pressure, heart rate, cardiac output, stroke volume, total peripheral resistance and left ventricular work increased and Pao 2 and hemoglobin saturation decreased. Arterial oxygen tensions below 80 torr were common at moderate and deep anesthetic levels of halothane plus 75% N 2O (H75N 2O). The specific contribution of N 2O, hypoxemia, hypercapnia, or temporal recovery (or a combination of these) in producing cardiovascular stimulation were not determined.
AB - The cardiovascular effects of equipoxent (minimum alveolar concentration) doses of halothane versus halothane plus 25% N 2O (H25N 2O) in spontaneously breathing dogs do not differ except that nitrous oxide increased mean arterial pressure (AP) and decreased arterial oxygen partial pressure (Pao 2). When 75% nitrous oxide was added to halothane anesthesia, AP, mean pulmonary artery pressure, heart rate, cardiac output, stroke volume, total peripheral resistance and left ventricular work increased and Pao 2 and hemoglobin saturation decreased. Arterial oxygen tensions below 80 torr were common at moderate and deep anesthetic levels of halothane plus 75% N 2O (H75N 2O). The specific contribution of N 2O, hypoxemia, hypercapnia, or temporal recovery (or a combination of these) in producing cardiovascular stimulation were not determined.
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UR - http://www.scopus.com/inward/citedby.url?scp=0016426302&partnerID=8YFLogxK
M3 - Article
C2 - 1111385
AN - SCOPUS:0016426302
VL - 36
SP - 197
EP - 200
JO - American Journal of Veterinary Research
JF - American Journal of Veterinary Research
SN - 0002-9645
IS - 2
ER -