Anterior ST-segment depression associated with acute inferior myocardial infarction: Clinical, hemodynamic, and angiographic correlates

John C Rutledge, Ezra A Amsterdam, H. Bogren, D. Arons

Research output: Contribution to journalArticle

6 Citations (Scopus)

Abstract

Clinical, hemodynamic, and angiographic data were analyzed in relation to the presence or absence of anterior (I, aVL, and V1-6) electrocardiographic ST-segment depression in 21 patients with acute inferior myocardial infarction. All data were obtained within 12 h of onset ofchest pain. Greater than 1 mm anterior ST-segment depression was frequent (19 of 21 patients) during the early phase of acute infarction. There was a significant correlation between the single electrocardiographic lead with the greatest inferior ST-segment elevation and the single anterior lead with the greatest ST-segment depression (r = 0.87, p < 0.001), and between the sum of ST-segment elevation in the inferior leads and the sum of ST-segment depression in the anterior leads (r = 0.77, p < 0.001). The patients with >2 mm anterior ST-segment depression had a higher left ventricular end-diastolic pressure than those with ≤2 mm ST-segment depression (18.4 vs. 10.8 mm Hg, p < 0.01) only when leads I and a VL were included, but there were no clinical, angiographic, or other hemodynamic differences between the groups. These findings do not correlate anterior ST-segment depression associated with inferior myocardial infarction with a higher cardiovascular risk with regard to the variables examined.

Original languageEnglish (US)
Pages (from-to)290-295
Number of pages6
JournalAmerican Journal of Noninvasive Cardiology
Volume1
Issue number5
StatePublished - 1987

Fingerprint

Inferior Wall Myocardial Infarction
Hemodynamics
Infarction
Blood Pressure
Pain
Lead

ASJC Scopus subject areas

  • Cardiology and Cardiovascular Medicine

Cite this

@article{c281eb766d4b427488c59d3ab5b5cf66,
title = "Anterior ST-segment depression associated with acute inferior myocardial infarction: Clinical, hemodynamic, and angiographic correlates",
abstract = "Clinical, hemodynamic, and angiographic data were analyzed in relation to the presence or absence of anterior (I, aVL, and V1-6) electrocardiographic ST-segment depression in 21 patients with acute inferior myocardial infarction. All data were obtained within 12 h of onset ofchest pain. Greater than 1 mm anterior ST-segment depression was frequent (19 of 21 patients) during the early phase of acute infarction. There was a significant correlation between the single electrocardiographic lead with the greatest inferior ST-segment elevation and the single anterior lead with the greatest ST-segment depression (r = 0.87, p < 0.001), and between the sum of ST-segment elevation in the inferior leads and the sum of ST-segment depression in the anterior leads (r = 0.77, p < 0.001). The patients with >2 mm anterior ST-segment depression had a higher left ventricular end-diastolic pressure than those with ≤2 mm ST-segment depression (18.4 vs. 10.8 mm Hg, p < 0.01) only when leads I and a VL were included, but there were no clinical, angiographic, or other hemodynamic differences between the groups. These findings do not correlate anterior ST-segment depression associated with inferior myocardial infarction with a higher cardiovascular risk with regard to the variables examined.",
author = "Rutledge, {John C} and Amsterdam, {Ezra A} and H. Bogren and D. Arons",
year = "1987",
language = "English (US)",
volume = "1",
pages = "290--295",
journal = "American Journal of Noninvasive Cardiology",
issn = "0258-4425",
number = "5",

}

TY - JOUR

T1 - Anterior ST-segment depression associated with acute inferior myocardial infarction

T2 - Clinical, hemodynamic, and angiographic correlates

AU - Rutledge, John C

AU - Amsterdam, Ezra A

AU - Bogren, H.

AU - Arons, D.

PY - 1987

Y1 - 1987

N2 - Clinical, hemodynamic, and angiographic data were analyzed in relation to the presence or absence of anterior (I, aVL, and V1-6) electrocardiographic ST-segment depression in 21 patients with acute inferior myocardial infarction. All data were obtained within 12 h of onset ofchest pain. Greater than 1 mm anterior ST-segment depression was frequent (19 of 21 patients) during the early phase of acute infarction. There was a significant correlation between the single electrocardiographic lead with the greatest inferior ST-segment elevation and the single anterior lead with the greatest ST-segment depression (r = 0.87, p < 0.001), and between the sum of ST-segment elevation in the inferior leads and the sum of ST-segment depression in the anterior leads (r = 0.77, p < 0.001). The patients with >2 mm anterior ST-segment depression had a higher left ventricular end-diastolic pressure than those with ≤2 mm ST-segment depression (18.4 vs. 10.8 mm Hg, p < 0.01) only when leads I and a VL were included, but there were no clinical, angiographic, or other hemodynamic differences between the groups. These findings do not correlate anterior ST-segment depression associated with inferior myocardial infarction with a higher cardiovascular risk with regard to the variables examined.

AB - Clinical, hemodynamic, and angiographic data were analyzed in relation to the presence or absence of anterior (I, aVL, and V1-6) electrocardiographic ST-segment depression in 21 patients with acute inferior myocardial infarction. All data were obtained within 12 h of onset ofchest pain. Greater than 1 mm anterior ST-segment depression was frequent (19 of 21 patients) during the early phase of acute infarction. There was a significant correlation between the single electrocardiographic lead with the greatest inferior ST-segment elevation and the single anterior lead with the greatest ST-segment depression (r = 0.87, p < 0.001), and between the sum of ST-segment elevation in the inferior leads and the sum of ST-segment depression in the anterior leads (r = 0.77, p < 0.001). The patients with >2 mm anterior ST-segment depression had a higher left ventricular end-diastolic pressure than those with ≤2 mm ST-segment depression (18.4 vs. 10.8 mm Hg, p < 0.01) only when leads I and a VL were included, but there were no clinical, angiographic, or other hemodynamic differences between the groups. These findings do not correlate anterior ST-segment depression associated with inferior myocardial infarction with a higher cardiovascular risk with regard to the variables examined.

UR - http://www.scopus.com/inward/record.url?scp=0023609511&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=0023609511&partnerID=8YFLogxK

M3 - Article

AN - SCOPUS:0023609511

VL - 1

SP - 290

EP - 295

JO - American Journal of Noninvasive Cardiology

JF - American Journal of Noninvasive Cardiology

SN - 0258-4425

IS - 5

ER -