AKAP5 keeps L-type channels and NFAT on their toes

Manuel F Navedo; Johannes W Hell

doi:10.1016/j.celrep.2014.05.052

AKAP5 keeps L-type channels and NFAT on their toes

Manuel F Navedo, Johannes W Hell

Pharmacology

Research output: Contribution to journal › Article › peer-review

3 Scopus citations

Abstract

In this issue of Cell Reports, Murphy etal. and Dittmer etal. present exciting new insight into the regulation of Ca²⁺ influx via the L-type Ca²⁺ channel Ca_v1.2 and how increased Ca²⁺ influx translates into localized activation of the nuclear transcription factor NFAT upon depolarization in neurons.

Original language	English (US)
Pages (from-to)	1341-1342
Number of pages	2
Journal	Cell Reports
Volume	7
Issue number	5
DOIs	https://doi.org/10.1016/j.celrep.2014.05.052
State	Published - Jun 12 2014

ASJC Scopus subject areas

Biochemistry, Genetics and Molecular Biology(all)

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10.1016/j.celrep.2014.05.052

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title = "AKAP5 keeps L-type channels and NFAT on their toes",

abstract = "In this issue of Cell Reports, Murphy etal. and Dittmer etal. present exciting new insight into the regulation of Ca2+ influx via the L-type Ca2+ channel Cav1.2 and how increased Ca2+ influx translates into localized activation of the nuclear transcription factor NFAT upon depolarization in neurons.",

author = "Navedo, {Manuel F} and Hell, {Johannes W}",

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AU - Hell, Johannes W

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AB - In this issue of Cell Reports, Murphy etal. and Dittmer etal. present exciting new insight into the regulation of Ca2+ influx via the L-type Ca2+ channel Cav1.2 and how increased Ca2+ influx translates into localized activation of the nuclear transcription factor NFAT upon depolarization in neurons.

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AKAP5 keeps L-type channels and NFAT on their toes

Abstract

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